AKI & CKD Nursing Guide 2026

⚕️ Medical Disclaimer: This content is for educational purposes only and is intended for licensed healthcare professionals. It does not constitute medical advice and should not replace clinical judgment, facility protocols, or physician orders. Always verify medications, doses, and procedures with your institution's guidelines.

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KDIGO AKI staging, prerenal vs intrinsic vs postrenal classification, FENa interpretation, CKD staging, uremic symptoms, electrolyte management, and dialysis indications.

1. AKI KDIGO Staging

StageSerum CreatinineUrine Output
Stage 1Rise ≥0.3 mg/dL within 48h OR 1.5–1.9× baseline within 7 days<0.5 mL/kg/hr for 6–12 hours
Stage 22.0–2.9× baseline<0.5 mL/kg/hr for ≥12 hours
Stage 3≥3.0× baseline OR ≥4.0 mg/dL OR initiation of RRT<0.3 mL/kg/hr for ≥24h OR anuria ≥12h

2. AKI Classification: Pre/Intra/Post Renal

CategoryCausesBUN/Cr RatioFENaUrine OsmolalityKey Feature
Prerenal (decreased perfusion)Hypovolemia (dehydration, hemorrhage, burns), CHF, hepatorenal syndrome, sepsis, NSAIDs, ACE inhibitors>20:1 (BUN disproportionately high)<1%>500 mOsm/kg (concentrated — kidneys trying to hold water)Kidneys intact but underperfused; responds to fluids
Intrinsic/IntrarenalATN (most common: ischemia, nephrotoxins — aminoglycosides, contrast, myoglobin); glomerulonephritis; interstitial nephritis (medications)<20:1>2% (damaged tubules can't reabsorb Na)<350 mOsm/kg (isosthenuria — kidneys losing concentrating ability)Muddy brown casts (ATN); RBC casts (GN); WBC casts (interstitial nephritis)
Postrenal (obstruction)BPH, prostate cancer, cervical cancer, bilateral ureteral stones, foley kinked/blocked, neurogenic bladderVariableVariableVariableAnuria more common; bilateral obstruction required for AKI (one functional kidney enough); bladder scan first

FENa Calculation

FENa (Fractional Excretion of Sodium) = (Urine Na × Plasma Cr) / (Plasma Na × Urine Cr) × 100

FENa <1% = Prerenal (kidneys avidly reabsorbing sodium to preserve volume)
FENa >2% = Intrinsic/ATN (damaged tubules cannot reabsorb sodium)
FENa 1–2% = Indeterminate (can occur with early ATN, contrast nephropathy, myoglobinuria)

Important: FENa is INVALID if patient has received diuretics (furosemide forces Na excretion regardless of volume status → falsely high FENa). Use FEUrea instead in diuretic-treated patients (FEUrea <35% = prerenal).

3. Common Nephrotoxins — Know What to Avoid

CategoryExamplesNursing Action
AminoglycosidesGentamicin, tobramycin, amikacinTrough-based dosing or extended-interval dosing; monitor Cr daily; hydrate well; avoid with other nephrotoxins
IV Contrast (iodinated)CT contrast, angiography dyePre-hydrate with IV NS; consider N-acetylcysteine; hold metformin 48h post-contrast; avoid in Cr >1.5–2.0 or GFR <30–45
NSAIDsIbuprofen, naproxen, ketorolacAvoid in AKI, hypovolemia, elderly, CKD, CHF, cirrhosis; afferent arteriole prostaglandin-dependent in these patients
ACE inhibitors/ARBsLisinopril, losartanHold in acute AKI and hypovolemia; efferent arteriole dilation → drops GFR when perfusion low; restart when stable
Myoglobin (rhabdomyolysis)Crush injury, severe exercise, statin toxicity, malignant hyperthermiaAggressive IV fluids (target UO >200 mL/hr until urine clears); sodium bicarbonate (alkalinize urine); CK levels; tea-colored urine; troponin may be falsely elevated
VancomycinIV vancomycinAUC-based dosing preferred over trough only (AUC 400–600 mg·h/L); monitor Cr and troughs; avoid concurrent nephrotoxins

4. CKD Staging (KDIGO 2012)

StageGFR (mL/min/1.73m²)DescriptionKey Management
G1≥90Normal GFR + kidney damage markers (proteinuria, hematuria)Treat underlying cause; BP control; RAAS blockade if proteinuria
G260–89Mildly decreasedMonitor; cardiovascular risk reduction
G3a45–59Mild-moderate decreaseAvoid nephrotoxins; adjust drug doses; anemia of CKD begins; phosphate restriction
G3b30–44Moderate-severe decreaseNephrology referral; EPO/ESA for anemia; vitamin D supplementation; dietary restrictions
G415–29Severely decreasedPrepare for renal replacement therapy; AV fistula planning (created now — takes 4–6 weeks to mature)
G5 (ESRD)<15 (or dialysis)Kidney failureDialysis or transplant

5. Uremic Symptoms

Uremia = accumulation of uremic toxins (BUN, creatinine, and other metabolic waste) in the blood. Occurs in advanced AKI (Stage 3) and CKD (G4–G5).

Signs and Symptoms (Uremia = AK PISS MAN):
A — Anemia (decreased EPO production)
K — K+ hyperkalemia (retention)
P — Pericarditis (uremic — friction rub; pericardial effusion risk)
I — Itching (uremic pruritus — from phosphate deposition; scratch marks on skin)
S — Seizures (severe uremia → encephalopathy → seizures)
S — Sensorium change (uremic encephalopathy: asterixis, confusion, stupor)
M — Metabolic acidosis (kidney can't excrete acid)
A — Anorexia, nausea, vomiting
N — Neuropathy (uremic peripheral neuropathy; restless leg syndrome)

Uremic frost: White crystalline deposits on skin (BUN >200 mg/dL) — rare now with early dialysis

6. Dialysis Indications (AEIOU)

Emergent dialysis indications — AEIOU:
A — Acidosis (metabolic acidosis pH <7.1–7.2 refractory to bicarbonate)
E — Electrolytes (hyperkalemia refractory to medical management)
I — Intoxication (dialyzable toxins: methanol, ethylene glycol, lithium, salicylates, theophylline)
O — Overload (fluid overload refractory to diuretics; pulmonary edema)
U — Uremia (BUN >100–150 with symptoms; uremic pericarditis; uremic encephalopathy)
NCLEX High-Yield: AKI & CKD

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