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SVT at the Bedside: Vagal Maneuvers, Adenosine, and the Pause That Scares Everyone

⚕️ Medical Disclaimer: This content is for educational purposes only and is intended for licensed healthcare professionals. It does not constitute medical advice and should not replace clinical judgment, facility protocols, or physician orders. Always verify medications, doses, and procedures with your institution's guidelines.

Part of the ICU Pharmacology Hub — browse every related guide in one place.

This article was created with AI assistance.

Updated July 2026  |  More ICU clinical guides →

The monitor alarms and the rate reads 180. The QRS is narrow, the rhythm is dead regular, and the patient is anxious, palpitating, maybe a little short of breath but talking to you. This is the classic picture of supraventricular tachycardia — an umbrella term for regular, fast rhythms that originate at or above the AV node, most commonly AV nodal re-entrant tachycardia (AVNRT). SVT is one of the few ICU rhythms where a bedside nurse, working within orders and protocol, moves through a fast, well-defined sequence: recognize it, decide if the patient is stable, try a vagal maneuver, and if that fails, give adenosine correctly. This guide walks that sequence and the physiology behind each step.

The short version: Regular, narrow-complex, fast (often 150–220), with P waves buried or absent = think SVT. Unstable (hypotension, altered mentation, ischemic chest pain, acute heart failure) → synchronized cardioversion, not drugs. Stable → vagal maneuver first, then adenosine 6 mg rapid IV push with an immediate 20 mL flush, escalating to 12 mg. Warn the patient and yourself: adenosine causes a brief, alarming pause before the rhythm resets.

Recognizing SVT — and the one imposter you must respect

SVT is regular, narrow (QRS < 0.12 s in the typical case), and fast enough that P waves are usually lost in the preceding T wave. Rates commonly sit between 150 and 220. Two look-alikes deserve a mention. The first is sinus tachycardia, which has a clear cause (fever, pain, hypovolemia, anxiety, sepsis) and a visible P before every QRS — you treat the cause, not the rate. The second, and the dangerous one, is atrial fibrillation with a rapid ventricular response, which is irregular — if you look carefully at the R-to-R intervals and they march unevenly, this is not AVNRT and adenosine is not the tool. And any wide-complex regular tachycardia should be treated as ventricular tachycardia until proven otherwise, not assumed to be "SVT with aberrancy."

Stable or unstable? This is the fork in the road

Before any maneuver or drug, answer one question: is the tachycardia causing hemodynamic instability? Signs of instability include hypotension, acutely altered mental status, signs of shock, ischemic chest discomfort, or acute heart failure — and a plausible link to the rate. An unstable patient in SVT does not get a slow trial of vagal maneuvers and drugs; they get prompt synchronized cardioversion under provider direction. The stable patient — uncomfortable but perfusing — is the one who walks down the vagal-then-adenosine ladder.

Vagal maneuvers: free, first, and more effective when done right

Vagal maneuvers increase parasympathetic tone at the AV node, slowing conduction enough to interrupt the re-entry circuit. The classic Valsalva — bearing down against a closed glottis — works, but the modified Valsalva is meaningfully more effective: have the seated patient strain for about 15 seconds, then immediately lay them flat and passively raise their legs to 45 degrees for 15 seconds. The postural change augments the reflex and converts a larger share of patients. Carotid sinus massage is a physician maneuver and is avoided in patients with carotid bruits or a history of stroke. Whatever the maneuver, have the patient on the monitor and a rhythm strip running so you capture the conversion.

Adenosine: the drug whose whole personality is its half-life

Adenosine transiently blocks conduction through the AV node, breaking the re-entry loop and letting the sinus node retake control. Its half-life is measured in seconds — under 10 — which drives everything about how it is given. Because it is destroyed almost the instant it hits the bloodstream, it must reach the heart in one fast bolus.

StepWhat to doWhy
AccessUse the largest, most proximal IV — an antecubital or a central line is idealThe drug must reach the AV node before it is metabolized
First dose6 mg rapid IV pushA slow push simply disappears before it works
FlushImmediate 20 mL saline flush, often via a stopcock or the two-syringe techniqueChases the tiny drug volume into the central circulation
EscalateIf no conversion, 12 mg rapid push + flush; a second 12 mg may follow per protocolRe-entry circuits vary in how much AV block they need
Warn the patient — and yourself — about the pause. Adenosine produces a short run of asystole or high-grade block before the rhythm resets. The patient often feels a wave of chest heaviness, flushing, or a sense of impending doom that lasts only seconds. Tell them it will pass quickly. Have a rhythm strip running, resuscitation equipment at hand, and defibrillator pads available — the pause is expected, but you are set up as if it were not.

Cautions worth carrying in your head

Adenosine can precipitate bronchospasm, so it is used cautiously in severe asthma or COPD. Its effect is prolonged by dipyridamole and by carbamazepine, and blunted by methylxanthines like theophylline and caffeine, which may demand higher doses. Patients with a transplanted (denervated) heart are exquisitely sensitive and dosing is reduced. And critically: if the "SVT" is actually pre-excited atrial fibrillation (Wolff-Parkinson-White with an accessory pathway), AV-nodal blockers including adenosine can dangerously accelerate conduction down the bypass tract — another reason that an irregular or wide, bizarre rhythm changes the plan.

Bottom line: Regular narrow-complex tachycardia in a stable patient is a maneuver-then-adenosine problem; in an unstable patient it is a cardioversion problem. Do the modified Valsalva properly, push adenosine fast into a big vein with an immediate flush, and set up for the alarming-but-brief pause before it converts. Confirm the rhythm is regular first — an irregular RVR or a wide-complex rhythm sends you down a different road.

Where to go from here

Pair this with the atrial fibrillation with RVR guide for the irregular imposter, the wide-complex tachycardia guide for the rhythm you must not mistake for SVT, and the synchronized cardioversion guide for the unstable pathway.

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